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Extra info for Cerebrovascular Transport Mechanisms: International Congress of Neuropathology, Vienna, September 5–10, 1982

Example text

Referenees 1. Dempsey EW, Wisloeki GB (1955) An eleetron mieroseopie study of the blood-brain barrier of the rat, employing silver nitrate as a vital stain. J biophys bioehem Cytol 1: 245-256 2. Brightman MW, Reese TS (1969) Junetions between intimately apposed eelI membranes in the vertebrate brain. J Cell Biol 40: 648-677 3. Reese TS, Karnovska MJ (1967) Fine struetural loealization of a blood-brain barrier to exogenous peroxidase. J Cell Biol 34: 207-217 4. Brightman MW, Klatzko I, Olsson Y, Reese TS (1970) The blood-brain barrier to proteins under normal and pathologieal eonditions.

13. 14. 15. 16. 17. 18. 19. 20. Anders JJ, Brightman MW (1979) Assemblies of particles in the cell membranes of developing, mature and reactive astrocytes. J Neurocytol 8:777-795 Anders JJ, Brightman MW (1981) lnitial changes in astrocytic membranes at the site of injury to the central nervous system. Anatomical Record 199, 9A (Abstract) Beggs JL, Waggener JO (1976) Transendothelial vesicular transport of protein following compression injury to the spinal cord. Lab lnvest 34:428-439 Bodenheimer TS, Brightman MW (1968) A blood-brain barrier to peroxidase in capillar'ies surrounded by perivascular spaces, Am J Anat 122:249-267 Bordi C, Perrelet A (1978) Orthogonal arrays of particles in plasma membranes of the gastric parietal cell.

However, in one rat, for example, 12 out of 36 vessels surrounded by HRP exudate had a total of 15 clefts between contiguous endothelial eells. Usually, one or two clefts would be ineluded in a single thin seetion. All of the interjunetional pools along eaeh of t~~ye clefts had been penetrated, from luminal to abluminal end, by La . This complete penetration was discernible in single thin sections. Serial seetions of other more undulating elefts were necessary to follow the extent of penetration (47, ef 27).

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